Phases: inflammatory (days 1–7), proliferative (days 3–21), remodeling (weeks–months). Intrinsic (tenocyte) vs extrinsic (synovial/paratenon) healing; adhesion formation from extrinsic fibroblasts. Early controlled mobilization enhances tensile strength and reduces adhesions in flexor tendons. Suture techniques: core locking (e.g., 4–6 strand) + epitendinous running improves gap resistance. Rehab protocols: Kleinert, Duran (flexor); early active motion in selected repairs.
What is the primary characteristic of the inflammatory phase of tendon healing?
Which type of tendon healing is primarily facilitated by tenocytes?
Which rehabilitation protocol emphasizes early active motion in selected flexor tendon repairs?
What is the main advantage of early controlled mobilization after tendon repair?
In the proliferative phase of tendon healing, what is primarily synthesized?
Which suture technique is known to improve gap resistance in tendon repairs?
What is a potential consequence of extrinsic healing in tendon injuries?
Which factor is NOT typically associated with delayed tendon healing?
During which phase of tendon healing does collagen maturation primarily occur?
What role do proteoglycans play in tendon structure?
