The effect of polydeoxyribonucleotide prolotherapy on posterior tibial tendon dysfunction after ankle syndesmotic surgery: A case report.

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Abstract

[Indexed for MEDLINE] Conflict of interest statement: The authors have no conflicts of interest to disclose. 13. Cureus. 2025 May 31;17(5):e85151. doi: 10.7759/cureus.85151. eCollection 2025 May. Congenital Absence of the Navicular Bone: A Rare Cause of Adult-Acquired Flatfoot and Posterior Tibial Tendon Dysfunction. Alhashimi I(1), Al-Taie A(2), Alam S(1), Ibrahem Adam R(3)(4). Author information: (1)Radiology, Hamad Medical Corporation, Doha, QAT. (2)Radiology, Hamad General Hospital, Doha, QAT. (3)College of Medicine (CMED), Qatar University, Doha, QAT. (4)Musculoskeletal Radiology, Hamad Medical Corporation, Doha, QAT. The navicular bone is critical for foot biomechanics, stabilizing the midfoot through its articulation with the talus and cuneiform bones. Variations, including accessory ossicles, hypoplasia, and complete absence, can lead to altered foot function. We present the case of a 58-year-old female patient who experienced persistent left foot pain. Imaging revealed the congenital absence of the navicular bone, resulting in altered biomechanics, midfoot instability, flatfoot deformity, and secondary degenerative changes. Posterior tibial tendon dysfunction (PTTD), a leading cause of adult-acquired flatfoot, is exacerbated by navicular anomalies. Advanced imaging plays a critical role in diagnosis, with treatment ranging from conservative management to surgery, depending on severity. Copyright © 2025, Alhashimi et al. DOI: 10.7759/cureus.85151 PMCID: PMC12212618