World journal of orthopedics | 2016 | van Bergen CJ, van den Ende KI, Ten Brinke B, Eygendaal D
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17. Pediatr Radiol. 2025 Jun;55(7):1386-1402. doi: 10.1007/s00247-025-06259-6. Epub 2025 May 16. Osteochondritis dissecans in children: location-dependent differences (part II: ankle and elbow). Nguyen JC(1)(2), Kan JH(3), Patel VS(4)(5), Blankenbaker DG(6), Rubin DA(7), Shea KG(8), Nissen CW(9), Jaramillo D(10), Ganley TJ(11)(5). Author information: (1)Section of Musculoskeletal Imaging, Department of Radiology, Children's Hospital of Philadelphia, 3401 Civic Center Blvd, 19104, Philadelphia, PA, USA. nguyenj6@chop.edu. (2)Perelman School of Medicine, University of Pennsylvania, Philadelphia, USA. nguyenj6@chop.edu. (3)Texas Children's Hospital, Baylor College of Medicine, Houston, USA. (4)Section of Musculoskeletal Imaging, Department of Radiology, Children's Hospital of Philadelphia, 3401 Civic Center Blvd, 19104, Philadelphia, PA, USA. (5)Division of Orthopaedic Surgery, Children's Hospital of Philadelphia, Philadelphia, US. (6)Musculoskeletal Imaging and Intervention, Department of Radiology, University of Wisconsin School of Medicine and Public Health, Madison, USA. (7)Department of Radiology, New York University Grossman School of Medicine, New York, USA. (8)Department of Orthopaedic Surgery, Stanford University School of Medicine, Stanford, USA. (9)Department of Orthopaedic Surgery, University of Connecticut and the Bone and Joint Institute at Hartford Hospital, Hartford, CT, USA. (10)Department of Radiology, Hospital for Special Surgery, New York, USA. (11)Perelman School of Medicine, University of Pennsylvania, Philadelphia, USA. The classic terminology "osteochondritis dissecans (OCD)" describes a pathologic alteration, centered at the osteochondral junction, involving the subchondral bone and/or its cartilaginous precursor, with risk for lesion instability and disruption of adjacent articular cartilage. Among children and young adults, these sites of osteochondrosis can be a cause of chronic joint pain and are most often found within the knee, the ankle, and the elbow joints. While Part I of this review series focused on shared key definitions, pathophysiologic principles, and imaging considerations, as well as unique differences between lesions at different locations within the knee joint, the current Part II article is devoted to lesions that involve the ankle and elbow joints. Following the outline of the Part I article, an evidence-based literature review on location-specific pathophysiology, imaging considerations, findings of lesion instability, and treatment selection considerations will be discussed for lesions involving the talar dome, capitellum, and humeral trochlea. © 2025. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature. DOI: 10.1007/s00247-025-06259-6
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